mouse anti tomm20 (Proteintech)
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96
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Proteintech
mouse anti tomm20
Mouse Anti Tomm20, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 1337 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/mouse+anti+tomm20/TOM20+Antibody/pm41637987-69-10-14
Average 96 stars, based on 1337 article reviews
Mouse Anti Tomm20, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 1337 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/mouse+anti+tomm20/TOM20+Antibody/pm41637987-69-10-14
Average 96 stars, based on 1337 article reviews
mouse anti tomm20 - by Bioz Stars,
2026-10
96/100 stars
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Related Articles
Immunofluorescence:Article Title: KLHL20 alleviates high glucose-induced mitochondrial apoptosis in renal tubular cells by targeting DAPK1 for ubiquitination and degradation. Article Snippet: Diabetic kidney disease (DKD) is a leading cause of end-stage renal disease, driven by hyperglycemia-induced mitochondrial apoptosis in renal tubular epithelial cells.. Death-associated protein kinase 1 (DAPK1) is a key mediator of cell death, but its regulation in DKD remains unclear.. Here, we investigated the mechanisms underlying DAPK1 upregulation and its role in mitochondrial apoptosis under high glucose (HG) conditions in HK2 cells and db/db mouse models. Staining:Article Title: KLHL20 alleviates high glucose-induced mitochondrial apoptosis in renal tubular cells by targeting DAPK1 for ubiquitination and degradation. Article Snippet: Diabetic kidney disease (DKD) is a leading cause of end-stage renal disease, driven by hyperglycemia-induced mitochondrial apoptosis in renal tubular epithelial cells.. Death-associated protein kinase 1 (DAPK1) is a key mediator of cell death, but its regulation in DKD remains unclear.. Here, we investigated the mechanisms underlying DAPK1 upregulation and its role in mitochondrial apoptosis under high glucose (HG) conditions in HK2 cells and db/db mouse models. Incubation:Article Title: Physical exercise protects neurons from energy deficit and improves cognitive function by upregulating astrocytic Slc2a1 in Alzheimer's disease. Article Snippet: Alzheimer’s disease (AD) has been associated with impaired energy metabolism and neuronal mitochondrial dysfunction, which contribute to the development of anxiety-like behaviors and spatial memory deficits.. Astrocytes are recognized as a critical source of metabolites for neurons, supporting mitochondrial respiration.. Although physical exercise (PE) Jo ur na l P re -p ro of Journal Pre-proof has shown therapeutic potential in AD models, the molecular mechanisms linking PE to metabolic reprogramming remain elusive. |